1·Pulmonary Alveolar-Capillary Changes in Rats Exposed to 100% Oxygen.
置于100%氧气压力下实验小鼠的肺泡-毛细管的变化。
2·Objective: To investigate the therapy of pulmonary alveolar proteinosis (PAP).
目的：探讨肺泡蛋白质沉积症(pap)的治疗。
3·The utility model is applicable to the administration for bronchiole and pulmonary alveolar.
本实用新型可用于毛细支气管及肺泡的给药。
4·Objective To study the diagnosis and pathological manifestations of pulmonary alveolar proteinosis (PAP).
目的总结肺泡蛋白沉积症(pap)的病理特征及病理诊断要点。
5·Purpose: To explore the HRCT manifestations and the diagnostic value of Pulmonary alveolar proteinosis (PAP).
目的：探讨肺泡蛋白沉积症(pap)的HRCT表现及诊断价值。
6·The structure of type I, IIalveolar cells and pulmonary alveolar capillary endothelial were maintained normal.
型肺泡上皮细胞形态完整，数目增多，板层小体结构趋于正常。
7·Objective: To improve the understanding of the diagnosis and treatment of pulmonary alveolar proteinosis (PAP).
目的：总结肺泡蛋白沉积症(pap)患者的诊治情况，提高对本病的认识。
8·Objective: To study X-ray diagnosis, classification and differential diagnosis of pulmonary alveolar proteinosis (PAP).
目的：探讨肺泡蛋白沉积症(pap)的X线诊断、X线分型与鉴别诊断。
9·Main mechanism of ALI induced by LPS is that it directly damaged pulmonary alveolar epithelium and vascular endothelium.
LPS致ALI的机制主要是直接损伤肺泡上皮和血管内皮细胞。
10·Objective: To summarize and analyze the clinical manifestations of pulmonary alveolar proteinosis (PAP) and its treatment.
目的：总结肺泡蛋白沉积症(pap)的临床特征、诊断及治疗。